Journal of Carcinogenesis & Mutagenesis

Carcinogenesis & Mutagenesis Outbreaks the Innovations in the Field of Oncology

Alireza Heidari^* American International Standards Institute (AISI), USA
^*Correspondence: Dr. Alireza Heidari, American International Standards Institute (AISI), USA, Email:

Received: 19-Mar-2020 Published: 07-Apr-2020, DOI: 10.35248/2157-2518.20.11.e125

Editorial Note

Carcinogenesis, also called oncogenesis or tumorigenesis, is the formation of a cancer, whereby normal cells are transformed into cancer cells. Mutagenesis is a process by which the genetic information of an organism is changed, resulting in a mutation.

The Journal of Carcinogenesis & Mutagenesis is an open access, peer-reviewed international journal that publishes scientific articles related to all aspects of cancer, by including cancer cell structure, photocarcinogenesis, adenocarcinoma pancreas prognosis, cancer epidemiology, etc.

The current volume 11, issue 2 various aspects of cancer were discussed by the authors from different parts of the world. In the research article, Soto-Cruz I, et al. studied NKG2D receptor engages ligands such as MICA and MICB, which activates cytotoxicity in NK cells leading to the destruction of tumour cells [1].

Fujioka K, in their review article evaluated a different genetic susceptibility contribution in migraine without aura (MWOA) and migraine with aura (MWA) has been suggested in the study using a genetic risk score (GRS) [2].

Maryam reported a forensic case of Potential of Germline CDKN2A I49T as a Targetable Driver Mutation: Prolonged Control of Refractory Osteosarcoma with CDK4/6 Inhibitor in a Familial Cancer [3].

Gorish BMT, studied about various environmental factors discussed in this study, only cadmium contact has a consistent and strong effect as a risk factor for prostate cancer [4].

Bursac D, et al. reported a Patient with Primary T790M Mutation in Lung Adenocarcinoma Treated with Gefitinib as a First-Line and Osimertinib as a Second-Line Therapy [5].

Olsson H, briefly described the cyclic Stimulation of the Breast Epithelium the Key Hormonal Factor Behind Breast Cancer? [6]. Sinha PK, described a commonly held belief nowadays that cancer originates in tissue-specific adult stem cells [7].

REFERENCES

1. Soto-Cruz I, Carreón OZ, Islas FT, Gallegos VJL, Dehesa ZA, Steider BW, et al. MICA Regulates the Expression of DAP10 and Signals through an Independent PI3K Pathway in NKG2D Positive Cervical Cancer Cells. J Carcinog Mutagen. 2019;10(1):329.
2. Fujioka K. Propensity to the Vascular Smooth Muscle Cell Abnormality in Migraine without Aura and Vasospastic Angina along with a Genome-Wide Association Studies. J Carcinog Mutagen. 2019;10(2):334.
3. Tehrani OS, Abdulhaq H, Delozier CD. Potential of Germline CDKN2A I49T as a Targetable Driver Mutation: Prolonged Control of Refractory Osteosarcoma with CDK4/6 Inhibitor in a Familial Cancer. J Carcinog Mutagen. 2019;10(1): 331.
4. Gorish BMT, Ournasseir MEH, Shammat IM. Effect of Age, Geographical Affiliation and Environmental Factors on the Development of Prostate Cancer among Sudanese Patients. J Carcinog Mutagen. 2019;10(3):337.
5. Bursac D, Sarcev T, Sazdanic DV, Djukic N, Sekurus V, Petkov S, et al. Patient with Primary T790M Mutation in Lung Adenocarcinoma Treated with Gefitinib as a First-Line and Osimertinib as a Second-Line Therapy: A Case Report. J Carcinog Mutagen. 2019;10(3): 338.
6. Olsson H. Is Cyclic Stimulation of the Breast Epithelium the Key Hormonal Factor Behind Breast Cancer?. J Carcinog Mutagen. 2019;10(4):340.
7. Sinha PK. The Common Genesis of All Cancers. J Carcinog Mutagen. 2019;10: 342.