Shyam Sundar Nandi
Department of Cellular and Integrative Physiology
University of Nebraska Medical Center, USA
Shyam Sundar Nandi, PhD (Postdoctoral fellow at University of Nebraska Medical Center) has experience in the field of heart failure and diabetes. He has expertise in diabetic heart failure and autophagy. His special interest is on microRNAs, autophagy, mitophagy, mitochondrial quality control axis and mitophagic cardiomyopathy in diabetic heart failure. He also has experience on regulation of β-adrenergic signaling, hyperhomocystinemia-H2S signaling, and pathological remodeling in diabetic heart. Additionally, he has good knowledge in molecular biology, proteomics and imaging techniques both at in vivo and in vitro levels. He has also experienced with evaluation of cardiac dysfunction by using MRI, echocardiography and pressure-volume loop. He has been honored with GOLD MEDAL for standing 1st position in MS and late Kamala Bala Mallick Award, and Best Student Award for scoring highest marks in MS. He received Best Poster Presentation Award in “International Symposium on Molecular Signaling” in 2013 and “Morris Karmazyn Award” for best poster in translational medicine, in annual meeting of the international academy of cardiovascular sciences: North American section in 2015. He is also actively involved to serve the scientific community by reviewing manuscripts from reputed peer-reviewed journals in cell biology and cardiovascular disease “AJP- Heart and Circulatory Physiology”, “International Journal of Diabetes and Clinical Research”, “Cell Communication and Signaling”, “Nature Scientific Report”, “Theranostics”, “Cellular Physiology and Biochemistry”, “Biological Trace element Research”.
Diabetic cardiomyopathy & Heart Failure, MiRNAs ,Mito-miRs, Autophagic and mitophagic adaptations in heart failure, Mitochondrial dysfunction and dynamics in diabetic hearts, Mito-miRs regulating ROS and UCPs and mitochondrial export-import machinery in diabetic hearts, H2S-Hyperhomocysteinemia signaling in diabetic heart failure, miRNAs regulated beta-adrenergic drive in diabetic hearts and factors regulate cardiac senescence in diabetic heart.