Cardiovascular Pharmacology: Open Access

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Parsons XH (2014) The openness of pluripotent epigenome–defining the genomic integrity of stemness for regenerative medicine. Int J Cancer Ther Oncol 2: 20114.

Parsons XH (2016) Direct conversion of pluripotent human embryonic stem cells under defined culture conditions into human neuronal or cardiomyocyte cell therapy derivatives. Human Embryonic Stem Cell Protocols 299-318.

Parsons XH (2016) Journal of Regenerative Therapeutics Volume 1, Issue 1, October 2016, Pages 1–8. Journal of Regenerative Therapeutics. 1 : 1-8.

Parsons XH (2016) Human pluripotent stem cell-based PluriXcel technology platforms provide stem cell treatment development and manufacturing innovations for progressing to the clinic. J Regen Ther 1: 1-8.

Parsons XH (2013) The designation of human cardiac stem cell therapy products for human trials. J Clin Trial Cardiol 1: 02.

Parsons XH (2014) The openness of pluripotent epigenome–defining the genomic integrity of stemness for regenerative medicine. Int J Cancer Ther Oncol 2: 20114.

Parsons XH (2016) Direct conversion of pluripotent human embryonic stem cells under defined culture conditions into human neuronal or cardiomyocyte cell therapy derivatives. Human Embryonic Stem Cell Protocols 16: 299-318.

Brown SA, Nhola L, Herrmann J (2016) Cardiovascular Toxicities of Small Molecule TKIs: An Opportunity for Systems‐Based Approaches. Clinical Pharmacology & Therapeutics.

Yaniv Y, Ahmet I, Tsutsui K, Behar J, Moen JM, et al. (2016) Deterioration of autonomic neuronal receptor signaling and mechanisms intrinsic to heart pacemaker cells contribute to age‐associated alterations in heart rate variability in vivo. Aging cell 15: 716-724.

Yaniv Y, Ahmet I, Liu J, Lyashkov AE, Guiriba TR, et al. (2016) Synchronization of sinoatrial node pacemaker cell clocks and its.

Captur G, Karperien AL, Hughes AD, Francis DP, Moon JC (2016) The fractal heart [mdash] embracing mathematics in the cardiology clinic. Nature Reviews Cardiology.

Liu J, Sirenko S, Juhaszova M, Sollott SJ, Shukla S, et al (2014) Cardiovascular and Cerebrovascular Aging–New Mechanisms and Insights: Age-associated abnormalities of intrinsic automaticity of sinoatrial nodal cells are linked to deficient cAMP-PKA-Ca2+ signaling. American Journal of Physiology-Heart and Circulatory Physiology 306: H1385.

Yaniv Y, Lakatta EG (2015) The end effector of circadian heart rate variation: the sinoatrial node pacemaker cell. BMB reports 48: 677.

Yaniv Y, Lyashkov AE, Lakatta EG (2014) Impaired signaling intrinsic to sinoatrial node pacemaker cells affects heart rate variability during cardiac disease. Journal of clinical trials 4.

Yaniv Y, Tsutsui K, Lakatta EG (2015) Potential effects of intrinsic heart pacemaker cell mechanisms on dysrhythmic cardiac action potential firing. Remodeling of cardiac passive electrical properties and susceptibility to ventricular and atrial arrhythmias 23: 9.

Yaniv Y, Lyashkov AE, Sirenko S, Okamoto Y, Guiriba TR, et al (2014) Stochasticity intrinsic to coupled-clock mechanisms underlies beat-to-beat variability of spontaneous action potential firing in sinoatrial node pacemaker cells. Journal of molecular and cellular cardiology 77: 1-3.

Liu J, Sirenko S, Juhaszova M, Sollott SJ, Shukla S, et al (2014) Age-associated abnormalities of intrinsic automaticity of sinoatrial nodal cells are linked to deficient cAMP-PKA-Ca2+ signaling. American Journal of Physiology-Heart and Circulatory Physiology 14:88.